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SPEECH BY DR. ROBERT STRECKER, M.D. ON THE AIDS VIRUS (given appx. Aug. 90)(posted to alt.conspiracy by Lance Bledsoe, July 8, 1993)It’s my pleasure to be here today and I’m glad that all of you could attend. We’re going to talk a little bit about AIDS. This is a topic we’ve all heard a great deal about, but quite frankly, most of what you’ve heard about is totally fiction. We got involved in this topic totally accidentally.In about 1983 my brother and I began working on an insurance plan for a large bank in southern California and in doing that in 1983, nobody knew what AIDS was going to cost us. So we decided that in writing an insurance plan for about 30,000 people, we would determine the cost of AIDS for ourselves. And that led us into this whole problem of not only AIDS, but what has come out about it. And basically what came out about the inquiry was the fact that we concluded that the AIDS virus was a man-made phenomenon. It’s not something that came from the jungles of Africa or some monkey virus, or from some monkey biting some African on the ass, and them BAM, 100,000 cases of AIDS come out of Africa.This virus was invented in a laboratory. They’d been working on producing it for 30-40 years. So, you would sort of have to ask yourself, when AIDS occurred, after they had been writing about it for such a long period of time, and they’ve been trying to make it for such a long period of time, why noone is running around, shouting and saying, “Hey, you know we finally did what we were attempting to do. We succeeded in producing a virus that destroys the immune system and kills people.” If people understood that this virus was designed in an attempt as an agent of mass destruction, instead of the misconception that somehow this is a virus that only attacks homosexuals or this is a virus that attacks drug abusers, I think we would all have a different appreciation.

There are two topics that most of us don’t really have any knowledge about. One is the precedence of these kinds of experiments. Recently in the Los Angeles Times just a couple weeks ago, I was reading an article where they were talking about a small town in Washington state, where a lot of people in that community had developed cancers of some strange sort. And of course the guys of whom all in their family developed these cancers out of the blue, happened to be living next to some kind of nuclear plant. The question was — was this plant somehow causing these cancers? Well, of course they denied it and the doctors all said, “These guys are crazy. These just happen appen to be occurring.”

But the fact was, as it came out, this nuclear plant had actually been releasing radiation into the atmosphere. This was probably not an accidental release, but an intentional release, and that they were actually studying the affects of these radiation releases upon the surrounding population. So you begin to get an appreciation of what the government is capable of doing.

Then you turn to the history of the CDC (Center for Disease Control) and the history of experimentation on the United States citizens without anybody’s knowledge or concern. Now, in a series of books such as A Higher Form of Killing by Packsman & Harris or Clouds of Secrecy by Leonard Cole, who is a Rutger’s professor, you can document over 300 open air biological experiments conducted on us without our consent or knowledge. That’s 300 that we have documented in the last 20 or 30 years. You have to only turn to the history of the Tuskegee, Alabama experiment to see the illustration of doctors neglecting what’s been going on. They’re saying, “Well, this kind of experiment couldn’t occur; we’d all know about it. Doctors would do something about it.” In Tuskegee, Alabama in the 30’s, black men were recruited for an experiment in which they were followed for about 40 years in open medical literature, where they was observed the progression in these black men of syphilis. Now, the interesting part about this experiment was, when penicillin became available, these men were prevented from being treated. This was conducted by the United States Public Health Service Department, which of course is now known as The Center for Disease Control in Atlanta, George, who are the captains in charge of what’s happening in our AIDS war. So you see, there is precedent for experimentation on people without anybody’s knowledge or consent.

There is also an entire history of experimentation on people with chemical agents, not only biological agents, but chemical agents, particularly in some experimental projects called MK-Ultra and some others where the United States Government Officials were actually introducing chemicals into men. For instance, in San Francisco, the most unusual that has been documented that is public, is where prostitutes were given LSD that they were giving to their “johns” or their sexual partners, and then the CIA were actually sitting behind one-way mirrors, and they were filming what was happening to these men who had been drugged with LSD from prostitutes. Of interest in the case is the prostitutes were arrested, then the CIA was actually intervening to see that they were released. So, this is some of the history of what’s gone on in the past and that is readily available for anyone to read about in some of the books that are out, A Higher Form of Killing again by Packsman & Harris or Clouds of Secrecy by Leonard Cole. The Killing Winds by Gene McDermott is another, so there is plenty of precedence. What about prediction? We talk about prediction; in other words in our review of the literature, we spent about 6 years in the library reading, digging out this stuff, and what we ran across was in 1975 in Tokyo at an International Assembly of Leukemia Experts was a guy named Jay Clemenson. Clemenson is still alive; this is an interesting story. In 1975, Jay Clemenson, who is a world-renowned Epidemiologist, was speaking before a group like this in Tokyo, all of whom were cancer and leukemia experts, got up and said the following, “We are in fact establishing conditions for a pandemic spread of an oncogenic virus varying on the scale of influenza of 1918.” Now, what is he saying? What he is saying is get ready, world, because scientists are making viruses that will cause leukemia or cancer, and that will spread as readily as the flu and kill more people than the influenza pandemic of 1918, which killed 1/5 to 1/3 of the world population. Now, that is exactly what has occurred today. That is *exactly* what has occurred. The existence of AIDS and its close relatives, is exactly the appearance of viruses that cause leukemia or cancer, that are spread in a sense like the flu, in fact AIDS is not spread as easily as the flu, but some of its other relatives probably are and these viruses are capable of killing not only (AIDS alone is capable of killing the entire human species) but certainly in concert all of them are capable of killing us off, totally annihilate the human race, and yet nothing is being done, more or less, where we are all sort of sitting around wondering what’s going on. There has been really no attempt made to control the epidemic of this disease.

Actually, I’m just finding it interesting, because if you want to learn about AIDS, the people that you talk to are the veterinarians. That’s why it was interesting when [it was] mentioned that the public health official here had talked about cattle. These diseases came from cattle and sheep. They didn’t come from monkeys in Africa. The story gets more preposterous as we get into it, but the reason that you know they didn’t come from monkeys, is because of the company that AIDS keeps, not because of the AIDS virus itself. If there were only AIDS virus, you probably couldn’t make any conclusion, because it could have appeared “out of the blue,” so to speak. Things do, in a sense, evolve or generate, but the interesting part is that if you look at the history of the theory of evolution and spontaneous generation, the United States National Institute of Health has spent billions of dollars telling us that spontaneous generation does not exist, and that evolution is the name of the game. And yet, when you come to the AIDS virus, it’s as if “spoof,” there’s no more history of evolution, now we have spontaneous generation, because it just “popped up.” It’s here, there is noone discussing where this virus came from. What was its evolution? What is its genesis? How is it put together? That’s how we got into this topic in looking at where did this thing come from? And when you look at the nature of the AIDS virus, what you will discover is something very interesting. The genes of the AIDS virus don’t exist in primates or man. If you took the genetic material of monkeys, chimpanzees, human beings and rearranged it, you cannot make AIDS. The genes of the AIDS virus exists in two other viruses called retroviruses of cattle and sheep. One of them is named bovine leukemia virus of cattle, which is a T-cell leukemia producing agent, just like Clemenson was talking about. The other is visna virus, a brain-rotting virus of sheep, that has managed to infect about 75% of the sheep on the western ranges of the United States and the rest of the world.

In the 1950’s, [there was an outbreak of a] bovine leukemia virus in Europe, a guy named Mamaro, and whole bunch of other retrovirologists in Europe, were looking at cattle and said, “You know, there’s something strange going on with these cattle. They have a strange kind of disease.” And they knew it was some kind of viral problem, but they couldn’t get a control over it. What they did was the exact thing that [was] mentioned; they established a program actually called Call and Kill. So, only the animals that they concluded were infected were called out and exterminated. All across Europe, they had a plan of Call and Kill of all the animals that were infected, and as a result they slaughtered hundreds of thousands of animals. This led to the development of disease free herds.

In the United States, they did a very interesting experiment. If you look at the way we test for the AIDS virus in the blood, we test for the presence of an antibody to the virus, that’s the screening tests that’s done. They don’t actually check and test in general for the virus itself. If you donate blood at your local Red Cross or hospital, they take the serum out of the blood, and then they check the serum to see if there’s an antibody in that blood directed against the AIDS virus.

Now, in the 1970’s, early 70’s, late 60’s, they did an interesting experiment here in the United States in Ames, Iowa, conducted by a guy named Vandermatten and girl named Miller. So Miller and Vandermatten took chimpanzees and injected them with a virus named bovine leukemia virus, because the questions was, “Is this virus dangerous for human beings?” And what happened was that these chimpanzees produced antibodies against that virus. So what was their automatic conclusion? The conclusion was that because these animals made antibodies against this virus, this virus represented no threat to the animal. So, therefore there was no program of containment of bovine leukemia virus in the United States. As a result, about 15% of the cattle in this country are now infected with bovine leukemia virus. If you look at the death certificates in the areas of massive milk producing states, like Wisconsin, Iowa, and Nebraska, there’s a virtual explosion of T-cell leukemia among dairy farmers, because primarily they drink in my opinion, unpasteurized milk. The only thing that has prevented all of us from being infected with the T-cell leukemia virus is the fact that pasteurization of milk kills the virus.

Go back to 1969, a testimony before the Church Committee in Congress; the Department of Defense representatives requested 10 million dollars to produce new viruses that could selectively destroy the immune system. In 1972, a group of virologists writing in the Bulletin of the World Health Organization, said this, “Let’s make a virus that will selectively destroy the T-cell system of man.” They went further and said, “Also, let’s make a virus that will selectively destroy the B-cell system.” And they wrote down, in Volume 47, page 257, 1972 Bulletin of the World Health Organization, and said why they wanted to make these agents. They said, “We can use these agents to produce certain kinds of cancers and leukemias. It will allow us to make these diseases, it will allow us also to make what we call dissolving diseases,” which is part two of that same request for production. So, in 1972, a group of virologists said, “Let’s make AIDS.”

In 1975, Clemenson says that it’s coming, and in 1980, it’s here and everybody’s wandering around, scratching their heads, saying, “Geez, where did all this stuff come from?” Well, really, to me it seems quite simple. This virus was produced in a laboratory by the recombination or the mixing or the melting together or the mating of two viruses, one named bovine leukemia virus of cattle, and the other named visna virus of sheep.

Now, if you look at the disease in humans that are presently running, there is a whole lot more going on out there besides AIDS. There is an infectious T-cell leukemia virus named HTLV-1 which is human T-cell leukemia virus 1, which is a confectious agent, it looks like bovine leukemia virus, it causes the same kind of disease in humans as it does in cattle, and this virus is probably far more infectious than the AIDS virus. This virus has managed to infect 20-30% of southern Japan already. It is infiltrating into the Asian countries, and because they’re so densely populated…

We can worry about Japan all we want, but I can tell you this, that short of a cure for these diseases, in about another 10 years or so, the entire population of Japan will suddenly get leukemia and die, short of there being a cure for this problem, because this disease will spread in that group of people due to their density of population. The same thing will occur in China, Taipei, India and the rest of the Asiatic countries. HTLV-2 which is HTLV human T-cell leukemia virus 2, causes hairy-cell leukemia in humans, the same as the similar disease bovine visna virus, causes a very unusual leukemia, hairy-cell leukemia in cattle. So, these diseases do have analogous agents that they could have evolved from or that they were derived from that are present today in laboratories around the world.

Ask yourself, how do you take a virus of cattle and sheep and make it into a virus of humans? In 1971 and 1972, the scientists of the world got very interested in this group of viruses called retroviruses. Retro stands for the presence of an enzyme called reverse transcriptates. Now, that doesn’t mean anything to us, but what does it really mean? Here is a virus which is in an RNA form. Our genes are in a DNA form. In other words, there are two separate entities, the viruses on RNA form are different than your genetic material, which is DNA. Why were they interested specifically in these agents more than the fact that they could make ethnic and racially specific bio-weaponry was this, and this is the very nature of the virus that allow them to do this: The virus enters the cell, it changes its form from an RNA form into a DNA form, which is now like your genes. It inserts itself into your genetic material and then interacts with the genetic material that is here; it then leads to production of new virus. The fact that it inserts itself into your genetic material and then expresses itself, in other words, once its inserted, it can then act — that fact made it perfect for introducing genes into species. So scientists said, “This is how we will introduce genes and manipulate the genetic material of species.”

They had been looking for a mechanism, trying to fire DNA into cells. In everything you can think of they were trying to manipulate to genetic materials. Because in their thinking they can do this job better than anybody else. So, this virus became of interest, because it has that ability; it will introduce genetic material from the outside and once it’s introduced, it changes forever that species. Just as with humans, now we have modified our genes, in a sense, forever, or short of a way to wipe out this species of this strange virus that’s inserting itself into us.

In 1972, when they were monkeying around with this thing in the United States National Institute of Health, a guy named Stuart Aronson published a paper and he was working with a mouse retrovirus; what Aronson discovered was this — he put that mouse retrovirus into a human tissue culture plate and then he came back after it was packaged, in other words, it grew there several times, and what he discovered was that this mouse retrovirus would no longer grow in mice, it would now grow most efficiently in human tissue. So he had discovered how to make a cross species jump. In other words, how you change the virus of one species into the virus of another species. This is how you take bovine leukemia virus of cattle and change it into HTLV-1 human T-cell leukemia virus. You merely have to package or grow the virus in human tissue for a sufficient period of time and you now have human T-cell leukemia virus. If you take bovine visna virus, the original AIDS virus and grow that virus in human tissue, you now have human AIDS virus.

One of the things we didn’t understand initially (or at least it wasn’t clear to us) in 1983 and 1984 was: were these people intentionally deceiving people, the American public — you and I, or were they just stupid? Now, don’t ever believe that they’re stupid. We asked a question of several virologists, and one of the questions was, “Could you take bovine leukemia virus of cattle and visna virus of sheep and make an AIDS-like agent?” In 1983 and 1984 we called up one of the world’s leading retrovirologists, one of the leaders in our AIDS industry, and said, “Can you do that kind of an experiment? Could you make and AIDS-like agent?” This is typical; he said, “Who wants to know?” And I said, “I want to know. I’m Robert Strecker.” So, he said, “Well, looking deep in the darkest part of Africa, you might find a virus similar to AIDS. But you could never, ever (NO,NO,NO) do that kind of an experiment.”

Now, for some reason, we didn’t quite believe him. So we went to the library and we put into the med-line search, and anybody can do this, if you don’t believe this is possible, simply go to your local library and ask for a med-line search, we thought what we’d call a virus if it was live AIDS and we knew that it had bovine and visna characteristics, so we said, “Well, give us all the papers on visna bovine virus and then give us all the papers on bovine visna virus from 1950 to date. That was from 1950 to like 1984. And whammo! out they came. Out came papers on a virus name bovine visna virus which has the exact same shape as the AIDS virus, it has the exact same molecular weight, it had the exact genetic structure in a sense, it had the exact same magnesium dependency, which is relatively unique to this class of agents, it had the exact same capability of killing T-cells selectively and yet, in the cumulative knowledge of the world’s AIDS expert, this virus didn’t exist. Now, that’s a lie.

The same technique that you make human T-cell leukemia virus from bovine leukemia virus is how you make human AIDS virus from bovine visna virus. And you take that virus and grow it in human tissue. In 1978 a paper was published in which they were growing bovine visna virus in human tissue. And of course, that’s how you adopt that virus in human. In that paper in 1978, which was published in The Journal of General Virology, for anybody who’s interested, said, “Is it possible, might it be, could it be that this virus is capable of producing either malignancy or a slow-virus disease of humans?” And of course, what is the malignancy? The malignancy is Kaposi’s sarcoma, and of course the slow-virus disease of humans is AIDS.

So, there’s prediction, there’s precedence, there’s production, what’s left? Well, inoculation. So, what do we think really happened? What we think happened was, in 1972 when the United States National Institute of Health was funded with millions of dollars to prove once and for all that viruses cause cancer, of which of course they didn’t pre-1972, and the reason that we know that, is because cancer was never infectious before, but it is now. In 1972, we produced a group of viruses that will cause cancer basically in the laboratories around the world and then in our opinion, these viruses were probably tested. We think they were tested in large populations in Africa, which explains how you get 300 million Africans probably infected today. We think that the entire continent of Africa will be extinct within the next 10 to 15 years. Dr. William Kendall Douglas, who recently returned from Africa, says that already he set up a clinic for treatment in Africa, he says already in Africa, AIDS is so devastating that they are dying literally like flies. So, in the next 10 or 15 years, again for sure, this is absolutely true, the same as what we have predicted before here. You will be able to go to Africa and verify for yourself, if you’re stupid enough to go, whether or not there’s anyone left. And, there won’t be anyone left if our predictions are correct.

The epidemic in Africa could not have started from a single-point infection, in other words, the numbers infected are so great, that there had to be a mass inoculation at some point in the mid-70’s. What we think really happened was a group of scientists went to Africa and actually tested these agents in Africa.

Now, how did they appear in the United States? If you look at AIDS in the United States, AIDS didn’t come here as a black, heterosexual disease. How did AIDS appear here? It appeared in a very, select group. Young, white, male homosexuals between the ages of 20 and 40 who live in select cities, New York, San Francisco, Los Angeles, Chicago and St. Louis. In 1978, it appeared in New York and in 1980 in San Francisco. Now that epidemiology is exactly the same as the United States Hepatitis B Vaccine study. We think that the virus was introduced into the homosexuals in this country in that project. Could it have been Anton Dega, the alleged gay airline steward who flew for the Canadian airlines? Well, to look at that story as put out by Schultz, in his book The Band Played On, it gets a little preposterous. First of all, the epidemiology of AIDS of a gay steward who is flying for a Canadian airlines should have reflected the cities that they were flying to, and it doesn’t. More importantly, allegedly the CDC was monitoring the activities of this steward, and so you must ask yourself why did the United States Federal body whose purpose it is to allegedly stop the spread of disease, didn’t interdict in a person who’s knowingly going around and spreading a uniformly fatal infectious disease. So what was the failure to act? Furthermore, how could you postulate that this man only had sex in unique cities separated by thousands of miles?

If you look at the theory of the virus coming from Haiti to New York City, you must wonder what happened to the gays of Miami, New Orleans and Houston, which have very large gay populations, whom are just as likely if not more likely to go to Haiti than the gays of New York City, San Francisco or Los Angeles. To our knowledge to date, if you look at the epidemiology of AIDS in the United States still today, it corresponds to the Hepatitis Vaccine B Study. The major cities affected are still those where the Hepatitis B Study was conducted.

Now, what is happening today? The rule of thumb for virology and this disease’s retroviruses is this: for every case that you see, there will be 100 cases coming. For every overt case of disease (this is in animals), for every sick cow that you can see, you have 100 affected. So in the United States today, if we have 130,000 or 140,000 cases of AIDS, that should calculate to 13, 14 or 15 million infected. Even at a 50 to 1 ratio, you’re still talking about 6 1/2 to 7 million people infected. If you calculate even 7 million people infected at a minimum cost of $100,000 a year, we’re talking about some astronomical sum being spent in the next 5 to 10 years just in the treatment of this disease alone. If you look at Africa, where there are already at least 2 million cases of overt disease, we’re talking about 200 million infected. So, that’s how we conclude that Africa won’t exist in the next 5 or 10 years. It will simply die out. You’ll see a population implosion.

The disease AIDS is already affecting Africa to such an extent that by satellite photos, the tropical forest in the African part of the African AIDS belt are already starting to re-grow. We talked to the representative of Uganda recently and he says this, “They have already written off everybody over 16.” Everybody over age 16, they are already considering is going to die, we concur, we agree. Their concentrating their educational efforts on everybody under 16 hoping that enough people will remain to keep the country, in a sense viable.

Now, what’s going to happen here? What we see happening here is more and more cases and the spread of this disease into the heterosexual population. This disease has nothing whatsoever to do with homosexuals in our opinion, except that’s where it was placed. In New York City already, AIDS is the #5 killer of women in the child-bearing age, and within 2 to 3 years, I can assure you that AIDS will be the #1 killer of women in the child-bearing age. Across this country within 3 to 5 years, AIDS will be the #1 killer of women of all groups in the child- bearing age.

What’s the solution? The solution is an even more interesting problem which we stumbled into in all of this sort of rambling about reading, and what we discovered that was, in our opinion, the disease can be fixed by a pulse electromagnetic wave, which led us into the theory of electromagnetic medicine, which led us to the theory of Raymond Roy Wright. The story of Wright is even more startling, because what if what Wright did is correct, and I believe that it was, then everybody who died of a cancer infectious diseases since 1920 died needlessly.

Wright’s theory is this, it’s very simple in principle. Just as with a crystal glass, if you radiate it with the right audiotone, what Wright said was that viruses and bacteria and cancers could be killed uniquely by a correctly pulsed electromagnetic radiation. And we believe that’s true, in fact, there’s an overwhelming amount of evidence that shows that that’s true. So we’ve got to redirect our research efforts away from the drugs, because quite frankly, I’m convinced that this disease and all of its relatives will never be cured by drug therapy because they’re far smarter than that. The disease is designed in a sense not to be cured by drugs. The disease was designed as an agent of mass destruction and it’s already done that.

Now, what can we do? Everybody asks that question. We’re going to get to that here, hopefully, we’re talking about what we can do politically, but this is a different sort of topic and I’m going to tell you some things that you can do. The first thing is, we have a videotape for sale back in the corner back there, so for any of you who don’t have that, or even if you don have it, you probably should pick up another copy and distribute it out to your local representatives or your senator, and you can’t simply send it to them. That doesn’t work; we tried that. You’ve got to go down and put it in their hands. The second thing is, we’ve got to become politically active and that’s what this is about. That’s why we’re meeting here and hopefully over the next 2,5,10 years, this change will occur. We’re already on a radio network called Sun Radio Network; we’re in about 100 cities nationwide, every night for 3 hours, 9:00 to 12:00 midnight PST. So if you’re in a city where you have the Sun Radio Network, talk to your radio station about the Dr. Raymond and Dr. Strecker show on, so we can be heard in your cities. Starting September 3 across the country in 200 and perhaps 400 cities, we’re going to be on CBN which is Christian Broadcasting Network, every morning at 9:00 PST. If you’re in a town where they have CBN Network, then go down to your radio station and start talking to them about picking up our show, which will be on 9:00 to 10:00. We’ll be answering medical questions, but we’ll also be dealing with other questions, such as this. In addition, we’re also writing now articles for about 10 newspapers already across the country. So what you can do, if you’re from a city, any size city, from 5,000, 50,000, 1,000,000 or 10,000,000, on Wednesday when you get home I want you to call my office at (213) 254-7127 and we’ll be happy to send you a press kit which you can take to your local newspaper and hopefully get us public in your local newspaper, so that people can be exposed to something other than the dribble that they’re being exposed to in the media.

The way that people do investigative reporting is they get a comment from somebody like us which differs from the standard comment. Then they get on the telephone and they call up a guy at the National Institute of Health who has a vested interest in none of this being known, because obviously if the United States general population had some idea that this virus was designed to kill humans specifically, there’d be a whole lot different approach to what’s going on in what we’ve been seeing. Also, there might be some oversight in some of the experiments that have been conducted in these laboratories. So, if we you can, take this press kit to your local newspaper and you promote getting us into that local newspaper as writing a medical column, which we’re already writing, we can show that we’re already writing a medical column for about 10 papers in California. Are we going to set up some kind of computer networking? (Yes.) Great. That’s another important thing. And computers will allow you access to this information. In the future, if you have interest, if they don’t provide machines for you, we’ll be able to provide machines for you. The thing about computers is that you’ll have instant access to information, not information that’s been exposed or filtered or whatever, and there will be more and more of what happens in the future is going to depend upon telecommunications upon accessed information about you educating yourself. The last thing that anybody in power wants is for the American public to become educated. That’s really where we’re headed.

So, the main thing that I want you to carry home from this conference is this:

1. The AIDS virus is not a happenstance occurrence of nature. This virus was produced specifically upon request and is designed to kill people.

2. This didn’t happen just by some accident. This has been worked on for about 30 to 40 years.

3. What happens in the future is going to be determined by what we do from this point on, in a sense. You’ve got to become involved politically; you’ve got to become involved actively, and that requires education of yourself and everyone around you, and you can’t be apathetic and sit at home on your couch and watch the boob tube, because you’re going to end up just as a boob.

We’ve got a couple of minutes left, any questions?

Q: Is the AIDS virus associated with Global 2000?

A: In the Global 2000, they talk about the mechanism of reducing the world’s population and a plan to reduce it by say 1/3 to 2/3. Actually, short of a mass terror, AIDS has already done that. It’s going to exterminate Africa and its relatives will exterminate all of Asia. And, remember this, this country is only about 5 years behind Africa. If you have, say 2 million people infected in the United States, if the disease doubles every year, or even every other year or every 2 years, 2 million, you only have to require about 6 to 8 doubling times before your reach the entire population of this country infected. And this disease is far more transmissible than just by sex. We haven’t gotten into that. That’s all on our videotape. We talk about how the disease is actually moved, we talk about why it’s not a sexually transmissible disease. We talk about a whole host of other factors that are interesting.

Q: What can we do to prevent catching the disease?

A: Right now, you can do this: You don’t use any IV drugs, you don’t have an excessive number of sexual partners, be monogamous if possible, and avoid any kind of biological agents that are being injected into you, no matter what they are.

Q: Will blue green algae build the immune system?

A: I’ll answer that by a quote from Dr. Gallo, who’s now under investigation for stealing the virus from the French, who is of course well known to all retrovirologists, but the United States has just gotten around to finally looking into that. Dr. Gallo said, “If you give AIDS to Superman and he’ll die of AIDS.” This disease is designed to destroy the immune system and it will do that, highly effectively.

Q: Why would the enemy want to create something like this?

A: We thought a great deal about that, and there is a theory of why biological agents have never been used. In other words, you’d have to vaccinate your population against this virus before you could use it. That’s not entirely true. In cattle, the disease has existed in cattle and sheep for many years. In cattle and sheep, there is a program we talked about already, called Call and Kill. You can control these diseases in animals, provided you’re willing to exterminate approximately 1% of your population every year. So, in a country like this, where obviously that kind of a program is not going to occur, and if we don’t enact some other kind of control mechanism or develop a cure, you could see the total annihilation of this country, whereas in a totalitarian state, if you were willing to exterminate 1% of your population a year, you would merely have to wait.

The motives. Again, it’s like the agenda of different groups. We use the analogy of nuclear power. If you look at Con Edison, they use nuclear power to generate electricity. If you look at the DOD, they use nuclear power to generate bombs. The agenda of this technique depends upon who you are. The cancer researchers were merely looking into the genesis of cancer, in other words, how does cancer occur and can we make viruses that will cause that, can we study it and make new diseases, can we study it and find a cure for cancer? Alternatively, other people, particularly bio-warfare people, were interested in producing ethnic and racially specific bio-weaponry which they can now do. In other words, if you tell me a select population that you want to exterminate, I could make for you a virus that would selectively kill that group. So again, the agenda depends upon who the group is. And it isn’t just us, it isn’t just the United States that’s been doing these sorts of experiments. These experiments are being conducted by all the industrialized nations of the world: Japan, France, England, Germany, Russia. This virus is being used in all the bio-warfare centers around the world.

Q: What technique can we use to prevent it or cure it, and is there any development of that?

A: No, there’s none, because the powers, the NIH, the people that are funding these programs are fixated upon, because of the incestuous nature between the United States National Institute of Health and the pharmaceutical industry, the incestuous nature has kept drug development the only answer, when in fact this disease in my opinion will only be cured in the long run by a physical process. That physical process or physical technique of pulse-electromagnetic radiation will cure not only all viruses but all bacteria and virtually every cancer.

There’s nothing available per se available to date that will uniquely prevent or cure this disease. We have been working on some techniques that are based upon very crude live technology that we may have available in the near future, but of course that could only be accepted outside of the United States and we’re working on getting that set up at the present time.

Q: What are the main methods of transmission of AIDS?

A: It is a cellularly transferred disease; it’s not transferred like any other disease that you’re aware of. It’s transferred by a large, white blood cell of the body. That cell carries the virus inside of it and that’s how the virus is moved between people. Now, the reason again that that’s not talked about, although the retrovirologists know that’s true, is because the only other model that exists in is visna and the reason they don’t want you talking about visna is because that takes you right back to bovine visna, right back into the laboratory, right back to the creation. There’s going to be an extended push towards trying to put this disease into monkeys. But yet how can a disease of monkeys that kills virtually 100% of those infected, why hasn’t it exterminated monkeys? Why hasn’t this disease been isolated from chimpanzees or monkeys before the mid-80’s or late 80’s when it was already in humans in the mid-70’s?

Q: Since this bovine virus apparently affects bovines, would it affect animals like goats and consequently their milk, and if you consumed that, would you be exposing yourself to that virus?

A: It already has. The way you make this disease run in another species is very simple, and that’s in fact how you make chimpanzee AIDS or monkey AIDS or cat AIDS or feline AIDS, if you notice there’s been a whole host of animal species that now have AIDS. It’s the same technique; you take the virus bovine visna, you grow it in the species that you want it to run in and you then have that species of AIDS. Now in goats particularly, there’s a very close relative of AIDS called cathlenarthritisencephalitis virus (CAEV), which will cause an arthritic condition of goats and which again is probably transferrable to humans if you drink unpasteurized goat milk.

Q: Can human body fluids transmit this virus?

A: Yes, if they’re infected with the macrophages primarily. The concentration of the virus is so small in human body fluids that it’s one of the interesting things. If you look at the concentration of the AIDS virus in semen, it’s virtually non-existent. If you look at the concentration of the AIDS virus saliva, it’s virtually non-existent. In fact, the concentration in saliva and semen are about the same. The concentration is at or below one free AIDS virus particle per milliliter. What does that mean? In a typical ejaculate from a male who’s infected with AIDS, you might have 3 species of semen. If you look at say syphilis or gonorrhea or herpes, in that 3 cc. of semen, you would find hundreds of thousands or organisms. That’s what allows the infection of the woman in this case, with either syphilis, gonorrhea or herpes. How many free AIDS virus particles are there in the same amount of semen? 3. Even more interestingly, if you look at the vagina itself, the only difference between the vagina and skin is that, instead of the fact that on your skin you have a dry layer on top, otherwise, they’re virtually the same. If you asked doctors how a virus gets inside of a cell, the answer is very interesting. They have a walking key phenomenon, in other words, the virus has like a little key on its surface and the cell has like the lock, and so the virus inserts the key into the lock and it turns the door lock and it opens the door and it goes in. That’s how viruses enter cells. It’s unique. They have on their surface little keys and they open the door. Now you say, is there a lock in the vagina, in other words, is there a receptor in the vagina? Well if a doctor tells you that there is (and luckily there isn’t) then you simply say, “Well gee doc, that means the whole human body is one giant AIDS receptor.” Because there is no difference between the mucosa of the vagina and the mucosa of your skin except for that dry layer. But the truth is, there isn’t any receptor for the virus in the vagina, so theoretically, if you painted a woman’s vagina with a pure concentration of AIDS virus, it couldn’t get in. So, you say, how does the virus get inside of a new person? This is how it gets inside. In that concentration, in the semen that’s ejaculated, there are these large mobile macrophages that are infected with the virus. Those macrophages, when the end up in the vaginal vault, they look outside and say, “It’s cold out here. I don’t like it out here. I’m going to go home.” And so, they have the ability to work their way through the wall, they actually separate the cells of the vagina, they’ll move apart the cells of the vagina, they’ll work their way through in an amoeboid action and they’ll close the wall behind them. Once they’re inside of her body, in this case a man who’s infected infecting a woman that’s not, what happens? What does her body say when it sees this macrophage coming in? It says, “An invader!” So her macrophages immediately surround that big killer cell and they punch holes all through it and inject it; and of course what are they injecting? They’re injecting the AIDS virus that’s contained inside of that cell. And so, that’s how the virus is transferred between people, even though there’s no virus present freely in the concentrated fluid itself.
This concludes Dr. Strecker’s speech.

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post 16-June-2007 18:03
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Interesting speech, but it’s still hard to find any support at all for Strecker, even today.

Sure would like to see some though. (I think?)

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post 17-June-2007 09:59
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Actually, there is possibly a surprising amount of support — and independant research – for the theories expressed by Dr. Strecker. Probably the most famous, as far as I’m aware, would be Len Horowitz. Here’s an article from his site,


A NEW THEORY ON THE ORIGIN OF AIDS (The following manuscript is a revised version of the scientific paper Dr. Leonard G. Horowitz and coauthors presented at the XI International Conference on AIDS in Vancouver, BC Canada on July 10, 1996. For more information contact 508-546-6586, and see “Emerging Viruses: AIDS & Ebola–Nature, Accident, or Intentional?” [Tetrahedron, LLC Press, 1996; $29.95] by Dr. Horowitz)


This article reviews scientific and U.S. Government documents that show AIDS-like viruses were developed by National Cancer Institute researchers along with military biological weapons contractors between 1962 and 1974 during the “Special Virus Cancer Program.” The possibility of this research giving rise to contaminated experimental hepatitis B (HB) vaccines, and thus, the simultaneous emergence of acquired immunodeficiency syndrome (AIDS) in New York City and Central Africa in 1978 is advanced.


Contrary to widespread speculations that human AIDS viruses arose from African green monkey viruses that naturally jumped species, in 1971, National Cancer Institute (NCI) researchers noted that “only one virus [of 27 then known retroviruses] which contains reverse transcriptase, does not seem to be oncogenic”, the simian foamy virus (SFV).(1)

Indeed, during the early days of cancer virus and viral vaccine research, simian viruses were common experimental contaminants. Could they have somehow mutated giving rise to the human immunodeficiency viruses (HIV-1 and 2) and AIDS?


The theory that viruses played a major role in carcinogenesis was most actively investigated by researchers at the NCI during the 1960s to mid-1970s. On the eve of Nixon’s “war on cancer,” NCI investigators explained how retrovirus related cancers such as lymphoma, leukaemia, and sarcoma might develop following virus infections. Twelve years later, in 1984, Dr. Robert Gallo and other esteemed NCI researchers advanced an essentially identical theory to explain AIDS.(1-10)

During the late 1960s and early 1970s, cell tumor biology researchers determined that synthetic RNA and feline leukaemia virus (FELV) “template” added to “human type C” viruses–those associated with cancers of the lymph nodes increased the rate of DNA production (and subsequent provirus and virus reproduction) as much as thirty times.(1) Such hybrid viruses, these researchers reported, may cause many cancers besides leukaemias and lymphomas, including sarcomas. Other NCI and Litton Bionetics teams reported modifying the fortieth discovered simian virus (SV40) by infusing it with nucleic acids from other species including FELV RNA, avian (i.e., chicken) myeloblastosis virus (AMV) RNA, associated with leukemia and sarcoma development, and mouse sarcoma RNA to: 1) make them carcinogenic, 2) prompt extreme immunosuppression in primates,(2,4,11) and 3) study RNA-dependent DNA polymerase (i.e., reverse transcriptase) and its relationship to human carcinogenesis,(6,11-14) For example, early work in viral engineering in relation to human carcinogenesis examined the activity of reverse transcriptase in normal versus acute immature leukaemic lymph cells (i.e., lymphoblasts). To do so, researchers evaluated the single stranded “70S RNA retrovirus” found in chickens which caused white bood cell (WBC) dysfunction, sarcomas, progressive wasting, and death–all prominent features of AIDS.(13)

Human WBCs were injected with this AMV RNA to determine if the cells were prompted to produce proteins and new viruses called for by the virogene.(14) Another team evaluated the human cancer-causing effects of the single-stranded 70S RNA reverse transcriptase enzyme. They used FELV and Mason-Pfizer monkey viruses to deliver these carcinogens to normal human lymphocytes.(15)

During parts of these experiments, NCI and Bionetics investigators even mixed RNA and DNA from chickens and cats with human WBC fractions including human lymphocyte DNA polymerases to induce cancer type and AIDS-virus-like reactions (see fig. 1).(15)

Other Gallo publications detailed the steps involved in creating immune-system-destroying cancer-causing viruses by adapting monkey, rat, and bird leukemia and tumor viruses for experimental use in a human (NC-37) cell line.16 One team discussed the synthesis of new RNA tumor viruses induced by 5-iodo-2′-deoxyuridine (IdU), a constituent of RNA in rodent cell cultures, and noted that chemical treatment might be used to halt the reverse transcriptase-linked viral reproduction cycle.(17)

In another report NCI researchers isolated a virus-like particle from human acute leukemic WBCs which had a specific density of 1.16-1.17g/ml, which allowed it to be repeatedly recovered without being destroyed by physical handling. Moreover, it was capable of producing the principal rapidly growing cancers seen in AIDS, including leukemias, sarcomas, and carcinomas.(19)

Defense Industry Interest

On April 4 and 5, 1969, at Fort Detrick, Maryland–America’s premier biological weapons testing facility, a controversial symposium on the entry and control of foreign nucleic acids into human cells was held.(10) That year the National Academy of Sciences-National Research Council informed Department of Defense (DOD) officials that “synthetic biological agents” that caused treatment resistant immunosupression could be developed “over the next five years” at a cost of $10 million.(9) A year later, NCI researchers described the experimental entry of bacterial RNA into human WBCs before a special symposium sponsored by the North Atlantic Treaty Organization (NATO). Their paper, published in the Proceedings of the National Academy of Sciences, discussed several possible mechanisms prompting the entry of foreign nucleic acids into lymphocytes.(2) Soon thereafter, the NCI acquired the lion’s share of Fort Detrick’s facilities.(10)

According to a 1970 Congressional Record, Bionetics Research Laboratories, a subsidiary of Litton Industries, Inc., was sixth on the list of U.S. Army biological weapons (BW) contractors.(20) Later Congressional Records showed that Bionetics’s affiliate–Litton Systems, Inc., another subsidiary of Litton Industries, Inc.–was among the most frequently contracted companies involved in BW research and development between 1960 and 1970.(21)

The Litton Industries, Inc. 1977-1978 annual reports stated,

“In June, [1976] Litton Bionetics won the fourth renewal of its contract to manage the operations of the National Cancer Institute’s Frederick (Md.) Cancer Research Center.”(22)

Later, Litton sold Bionetics Research Labs to Medpath–a subsidiary of Dow Corning–among the largest medical laboratories in the United States, yet continued to administer the lion’s share of NCI’s Frederick operations funding to the time of this writing.

Furthermore, NCI staff reports revealed that Litton Bionetics had been granted the service contract to supply all NCI researchers, worldwide, with virtually every primate cancer research material requested, including seed viruses and viral hybrids, experimental reagents, and colony born monkeys, including M. mulatta, associated with the major monkey AIDS virus outbreaks in California’s Davis Lab, and the 1967 Marburg virus outbreaks in three European vaccine production facilities and the African green C. aethiops.(24-26) Furthermore, from these publications, a list of the viruses and virus recombinants that Bionetics researchers developed, tested, and supplied to other NCI researchers during the 1960s and early 1970s was developed (see fig. 2).

A 1971 Litton Bionetics research report noted that “highest priority was given to the search for human leukemia viruses resembling the type-C viruses causing chicken and mouse leukemias” beginning as early as 1962.(23) Bionetics researchers, who received approximately $2 million annually for this work, reported:

“Several of the Type C viruses are established as the causative agents in leukemias, lymphomas, and sarcomas of chickens, mice, cats and hamsters. Many of these can infect and produce malignancies in other species (e.g., a sarcoma virus of the cat produces tumors in marmoset monkeys). Furthermore, some of these viruses can cause malignant transformation to occur in animal and human cells grown in the laboratory (e.g., cat leukemia and sarcoma viruses alter embryonic human cells). Type C virus particles have been found in association with malignancies of a spectrum of animal species including nonhuman primates, rats, cattle, wooley monkeys, gibbons, and man. . . .”(24)

Though some contemporary investigators have argued that HIV-1 and 2 are not type-C viruses,(27) more recently, researchers noted they go through a stage of type-C morphogenesis during replication and look and behave similar to the type-C viruses. “Although not classified as type-C viruses, lentiviruses follow a similar assembly strategy, by which capsid [shell] formation and budding [of the virus from the infected cell] occur simultaneously.”(28)

Perhaps not coincidentally, during the metamorphosis of HIV, researchers found “a reproducible peak of viral protein in the fraction corresponding to a density of approximately 1.15 to 1.16g/ml . . . in gradients of gag HIV,” that is, the gene that codes for the inner shell, capsid-like structure, of the AIDS virus.28 It may be recalled that Gallo et al. reported this number also, but in 1973, after repeatedly recovering the same density “virus-like particle” from human leukemic cells that was capable of producing the principal rapidly growing cancers seen in AIDS.19 Moreover, Kyle noted the United States Food and Drug Administration (FDA) Bureau of Biologics found a similar characteristic in the “adventitious virus” found in some live polio vaccine approved by and released in 1977.(29)

It is known that RNA viruses in general, and type-C and D lentiviruses in particular, “undergo extensive genetic variation as a result of error-prone replication and recombination such that they are considered to exist as ‘quasispecies,'” that is, a population of relatives with similar genes.30 One researcher noted “the exceptional ability of HIV-1 to mutate results in rapid development of quasispecies which evade host defenses and become resistant to various antiviral” agents.(31)

Bionetics/NCI researchers went on to report that, “Reactions between Type C viruses causing leukemias and sarcomas (solid tumors),” were a major area of interest for cancer prevention studies including the detection of cancer viruses, and viral vaccine experiments. The investigators wrote:

“When inoculated into appropriate cell cultures, type C sarcoma viruses of chickens, mice and cats produce foci [cancerous growths] of altered cells. This fundamental discovery provides a readily visible indicator reaction for the detection of sarcoma viruses. On the other hand, leukemia viruses grown in tissue culture do not cause foci or other detectable changes. The finding that leukemia viruses can either inhibit or enhance focus formation by sarcoma viruses of the same species has led to the development of methods for the detection and quantitation of leukemia viruses indirectly.

Certain of the chicken, cat and mouse sarcoma viruses are “defective” in that they do not produce foci in cell cultures or tumors in animals in the absence of a co-infecting, ‘helper’ leukemia virus. [Note the researchers called carcinogenic viruses “defective” if they were unable to produce cancers without the help of other factors including chemicals, radiation, and here leukemia viruses.] Further, in the presence of a defective sarcoma virus the helper action of leukemia viruses can be used as a specific indicator for their detection and quantitation. It is now believed that defective sarcoma virus leukemia virus interactions may be more widespread in nature than originally thought and that similar systems may be found in man. A mouse leukemia virus which has been adapted to grow in human cells is now available to search for defective human sarcoma viruses, if they exist.”(24)

In continuing this effort, they developed an “alternative approach” for the detection of possible human leukemia viruses that employed recombinant cat and mouse leukemia and sarcoma viruses engineered to cross species.

“A defective mouse sarcoma virus and its leukemia virus helper can be made to form tight functional aggregates, which behave as one virus. Using a mixture of mouse sarcoma virus and cat leukemia virus, a hybrid aggregate which could be grown continuously in cat cells was produced. [As Gallo et al. also reported.(14,15)] Because the aggregate is defective, it requires the simultaneous presence of a cat leukemia virus for producing altered foci in cat cells. Thus, a focus forming sarcoma virus of the mouse, artificially changed to one possessing infectivity for cat cells, can now be used in cultures for the detection of cat leukemia viruses.

This hybrid virus, as well as the cat leukemia virus, will also grow in human embryonic cells in tissue culture. If sufficient amounts of the Type C particles found in association with human leukemia can be obtained, the possibility exists that the cat-adapted mouse sarcoma virus can be hybridized with the human agent to produce an indicator system for the detection of human leukemia viruses. [Figure 3 presents a graphic description of this work.](24)

The NCI staff went on to explain their work elucidating: 1) the “biochemical pathways of tumor virus infection and replication,” 2) reverse transcriptase activity “in cells of patients with acute lymphoblastic leukemia . . . sarcomas, Burkitt’s lymphoma and breast cancer,” 3) experiments with Type B viruses thought to be associated with breast cancer, 4) Herpes-type viruses “associated with some forms of chronic leukemia, lymphoma, and postnasal carcinoma,” and 5) Epstein-Barr viruses extracted from Burkitt’s lymphomas and postnasal carcinomas. Vaccines, the NCI researchers explained, were expected to be developed from these efforts to help prevent and treat human cancers as coordinated “through the International Agency for Research on Cancer (IARC) in the West Nile District of Uganda.”(24)

A Possible Iatrogenic Cause of AIDS

In May 1942, George W. Merck was commissioned by President Franklin D. Roosevelt to direct the War Research Service overseeing America’s biological weapons industry.(32) Since then, the Merck company, in collaboration with the U.S. Public Health Service, provided ongoing expertise to the U.S. Army and its contractors “to bolster ongoing projects in fields in which it has an independent interest.”20

A service contract awarded Merck and Company, Inc., under the “Special Virus Cancer Program (SVCP),” called for “oncogenic virus research and vaccine development.” The chief objective of this work was reported as being “of fundamental importance to the goals of SVCP.” Their proposed course of study included �work towards development of a feline leukemia-sarcoma virus vaccine and a herpesvirus type 2 vaccine [to] be continued as rapidly as possible.”(33)

This grant description revealed that simian viruses (SV40)–currently suspected as being an AIDS virus progenitor29�and their “tumor cell ghosts” were prepared and used as principle carcinogenic triggers against which “non-protective SV40 tumor cell vaccines” were tested.33 This work was done at the same time Merck’s chief vaccine developer, tumor cell virologist Maurice Hilleman collaborated with Hepatitis B (HB) vaccine pioneer Dr. Saul Krugman of New York University Medical Center, another documented Army biological weapons contractor,(20) and Robert Purcell of the National Institute for Allergies and Infectious Diseases (NIAID) to develop and test the first “4 lots of vaccine that would amount to perhaps 200,000 human doses” by 1974.(34-36)

Bionetics military supplied rhesus monkeys and chimpanzees were used to develop these vaccines during this “initial limited clinical test for establishing safety and measuring antibody response [in human subjects].” This work was based on pilot investigations conducted between 1967 and 1971 with “heat-inactivated hepatitis B vaccine” in animals and high risk human subjects in New York and Central Africa.(34)

At this time Dr. A. M. Prince, charged with overseeing the Laboratory of Virology at the New York Blood Center, wherein the non-human primates were housed, reported a major biohazard and containment problem. Prince admitted, “I would say more than 70%” of the animals became environmentally infected with hepatitis B (and likely other viruses) during their captivity.(34)

In 1974, Purcell reported failed attempts to grow the HB seed viruses, needed for these vaccines, in cell cultures. Willowbrook State School (Staten Island, NY) mentally retarded children, rhesus monkeys, and chimpanzees, he announced, were successfully used instead to culture the viruses subsequently inoculated into high risk human subjects (e.g., Willowbrook children, Central African villagers, and apparently New York’s gay men as well) to develop the various vaccine subtypes.(35) “Cross-challenge experiments, and evaluation of various aspects of passive and active immunization against hepatitis B infection,” Purcell explained, then proceeded in collaboration with the FDA.

Thus, simian viruses, and/or Bionetics engineered viral hybrids, infecting chimpanzees during this work might have infected humans and given rise to HIV-1 or its immediate progenitor(s). As Shultz explained, “a lentivirus isolated from chimpanzees (SIVcpz)” is “the closest primate relative of HIV-1.”(27) Given, Bionetics’s involvement in primate cancer virus and animal supply to these New York/Bethesda/Uganda investigators, SIVcpz might have evolved because the chimps had likely been among the first creatures to be exposed to man-made retroviruses by way of direct inoculation or experimental monkey cohabitation.

It is also possible, even if Merck’s human experimental HB vaccine hadn�t included contaminated chimpanzee serum, only serum taken from New York’s children and/or gay men, live viruses injected around 1970 could have combined with the simian viruses (e.g., SV40, SIVagm, or SFV) the donors may have carried following vaccination with Merck’s polio vaccines administered during the previous decade.(29)

These facts provide additional background for evaluating Hilleman’s 1986 published comments of having imported AIDS into North America by way of African green monkeys destined for use in Merck’s viral and vaccine research. In an effort to reduce laboratory and vaccine contamination, Hilleman reported, “I brought African greens in. I didn’t know we were importing AIDS virus at the time.”(37)

Summary and Conclusions

This article reviews scientific literature and U. S. government documents that provide additional insight into the iatrogenic theory of AIDS. All it may have taken was one monkey used to develop the initial pilot HB vaccine lots, administered virtually simultaneously in New York City and Central Africa by 1974, carrying iatrogenically evolved or genetically engineered simian sarcoma-leukemia virus hybrids, to have started the AIDS epidemic.(38) This knowledge is important for at least three reasons: 1) the guilt and stigma attached to the victims of AIDS, homophobia, and racism, may ease in light of these findings; 2) new therapies might evolve from this knowledge; and 3) a thorough independent investigation and analysis of the aforementioned facts may help to prevent future outbreaks and epidemics.(39)

This work additionally supports others who have called for careful PCR analyses of suspected vaccine lots allegedly in safe keeping at the FDA.(38) Furthermore, as this report unearths evidence linking Willowbrook State School children, and apparently other high risk groups in New York City and Central Africa, to possibly contaminated experimental HB vaccines developed in chimpanzees, look-back studies of AIDS cases among those who received these early vaccines is clearly warranted.

1. Gallo RC, Sarin PS, Allen PT, Newton WA Priori ES, Bowen JM and Dmochowski L. Reverse transcriptase in type C virus particles of human origin. Nature New Biology 1971;232:140-142; see also Gallo RC. Transfer RNA and transfer RNA methylation in growing and “resting” adult and embyonic tissues and in various oncogenic systems. Cancer Research 1971;31:621-29.
2. Herrera F, Adamson RH and Gallo RC. Uptake of transfer ribonucleic acid by normal and leukemic cells. Proc Nat Acad Sci 1970;67;4:1943-1950. This paper was presented before NATO scientists at the “International Symposium on Uptake of Informative Molecules by Living Cells, Mol, Belgium, 1970,” the year in which $10 million in funds were appropriated by the Department of Defense for the development of AIDS-like viruses.
3. Gallo RC, Perry S and Breitman RT. The enzymatic mechanisms for deoxythymidine synthesis in human leukocytes. Journal of Biological Chemistry 1967;242;21:5059-5068.
4. Gallo RC and Perry S. Enzymatic abnormality in human leukaemia.Nature 1968;218:465-466.
5. Gallo RC and Breitman TR. The enzymatic mechanisms for deoxythymidine synthesis in human leukocytes: Inhibition of deoxythymidine phosphorylase by purines. Journal of Biological Chemistry 1968;243;19:4943-4951.
6. Gallo RC, Yang SS and Ting RC. RNA dependent DNA Polymerase of human acute leukaemic cells. Nature 1970;228:927-929.
7. Gallo RC and Longmore JL. Asparaginyl-tRNA and resistance of murine leukaemias to L-asparaginase. Nature 1970;227:1134-1136.
8. Horowitz LG. Deadly Innocence: The Kimberly Bergalis Case: Solving the Greatest Murder Mystery in the History of American Medicine.
Tetrahedron, LLC., 1994, p. 14.
9. Department of Defense Appropriations For 1970: Hearings Before A Subcommittee of the Committee on Appropriations House of Representatives, Ninety-first Congress, First Session, H.B. 15090, Part 5, Research, Development, Test and Evaluation, Dept. of the Army. U.S., July 1, 1969, Government Printing Office, Washington, D.C., pg. 79 and page 129 of supplemental record obtained through the Freedom of Information Act.
10. Washington Correspondent. Relief of Fort Detrick. Nature 1970;228:803; regarding controversial conference see: Boffey PM. Detrick birthday: Dipute flares over biological warfare center. Science April 19, 1968;171;285-288.
11. Gallaher RE, Ting RC and Gallo RC. A common change aspartyl-tRNA in polyoma and SV transformed cells. Biochimica Et Biophysica Acta 1972;272:568-582.
12. Fujioka S and Gallo RC. Aminoacyl transfer RNA profiles in human myeloma cells. Blood 1971;38;2:246-252.
13. Smith RG and Gallo RC. DNA-dependent DNA polymerases I and II from normal human-blood lymphocytes. Proceedings of the National Academy ofSciences 1972;69;10:2879-2884.
14. Bobrow SN, Smith RG, Reitz MS and Gallo RC. Stimulated normal human lymphocytes contain a ribonuclease-sensitive DNA polymerase distinct from viral RNA-directed DNA polymerase. Proceedings National Academy of Sciences 1972;69;11:3228-3232.
15. Robert MS, Smith RG, Gallo RC, Sarin PS and Abrell JW. Viral and cellular DNA polymerase: Comparison of activities with synthetic and
natural RNA templates. Science 1972;176:798-800.
16. Gallo RC, Abrell JW, Robert MS, Yang SS and Smith RG. Reversetranscriptase from Mason-Pfizer monkey tumor virus, avian myeloblastosis
virus, and Rauscher leukemia virus and its response to rifamycin derivatives. Journal of the National Cancer Institute 1972;48;4:1185-1189.
17. Wu AM, Ting RC, Paran M and Gallo RC. Cordycepin inhibits induction of murine leukovirus production by 5-iodo-2�-deoxyuridine. Proceedings of the National Academy of Sciences 1972;69;12:3820-3824.
18. Gillespie D, Gillespie S, Gallo RC, East J and Dmochowski L. Genetic origin of RD114 and other RNA tumor viruses assayed by molecular hybridization. Nature New Biology 1973;224:52-54.
19. Gallo RC, Miller NR, Saxinger WC and Gillespie D. Primate RNA Tumor Virus-Like DNA Synthesized Endogenously by RNA-Dependent DNA Polymerase in Virus-like Particles from Fresh Human Acute Leukemic Blood Cells. Proceedings National Academy of Sciences 1973;70;11:3219-3224.
20. Department of Defense Appropriations For 1970: Hearings Before A Subcommittee of the Committee on Appropriations House of Representatives, Ninety-first Congress, First Session, H.B. 15090, Part 5, Research, Development, Test and Evaluation, Dept. of the Army. U.S. Government Printing Office, Washington, D.C., 1969, p. 689; see also Congressional Record, August 8, 1969, p. 23073, and for U.S. Public Health Service involvement and funding see page 23079.
21. Committee on Human Resources, United States Senate. Hearings before the Subcommittee on Health and Scientific Research, Biological Testing Involving Human Subjects by the Department of Defense, 1977: Examination of Serious Deficiencies in the Defense Departments Efforts to Protect the Human Subjects of Drug Research. Washington, D.C.: U.S. Government Printing Office, May 8 and May 23, 1977, pp. 80-100.
22. Litton Industries, Inc. Annual Report[s] to the Securities and Exchange Commission for Fiscal Year Ended July 31, 1977 [and 1978]. Commission file number 1-3998. Securities and Exchange Commission, Office of Reports, October 31, 1977 [and October 30, 1978].
23. NCI staff. The Special Virus Cancer Program: Progress Report #8 [and #9].Office of the Associate Scientific Director for Viral Oncology (OASDVO). J. B. Moloney, Ed., Washington, D. C.: U. S. Government Printing Office, 1971 [and 1972]. Note: This is a very hard publication to find. Few library data bases have it listed, including the NCI Library at Fort Detrick. It is available through the Davis Library, The University of
North Carolina, Chapel Hill, Government Documents Department Depository, Reference # HE 20.3152:V81.
24. Ibid., 15-19; 20-26.
25. Ibid., 187-188; and in 1972 Progress Report #9, pp. 273-289.
26. Fine DL and Arthur LO. Prevalence of natural immunity to Type-D and
Type-C Retroviruses in primates. In: Viruses in Naturally Occurring
Cancers: Book B. Myron Essex, George Todaro and Harald zur Hausen, eds.,
Cold Spring Harbor, NY: Cold Spring Harbor Laboratory, 1980, Vol. 7,
pp. 793-813; see also Gallo RC, Wong-Staal F, Marhkam PD, Ruscetti R,
Kalyanaraman VS, Ceccherini-Nelli L, Favera RD, Josephs S, Miller NR
and Reitz, Jr MS. Recent studies with infectious primate retroviruses:
Hybridization to primate DNA and some biological effects on fresh
human blood leukocytes by simian sarcoma virus and Gibbon ape leukemia
virus. Ibid.. 793-813.
27. Shultz TF. Origin of AIDS (letter to the editor). The Lancet 1992;339:867.
28. Sakalian M, Parker SD, Weldon RA and Hunter E. Synthesis and assembly of retrovirus gag precursors into immature capsids in vitro.
Journal of Virology 1996;70;6:3706-3715.
29. Kyle WS. Simian retroviruses, poliovaccine, and origin of AIDS. The Lancet 1992;339:600-601; Personal communication from Walter Kyle, May 19, 1996.
30. Drew L, Lichtenstein, Issel CJ and Montelaro RC. Genomic quasispecies associated with the initiation of infection and disease in ponies
experimentally infected with equine infectious anemia virus. Journal of Virology 1996;70;6:3346-3354.
31. Shaheen F, Duan L, Zhu M, Bagasra O and Pomerantz RJ. Targeting human immunodeficiency virus type 1 reverse transcriptase by intracellular expression of single-chain variable fragments to inhibit early stages of the viral life cycle Journal of Virology 1996;70;6:3392-3400.
32. Covert NM. Cutting Edge: A history of Fort Detrick, Maryland 1943-1993. Fort Detrick: United States Army Garrison, 1993, pp. 17-19.
33. NCI staff. Op cit. p. 111; and in 1972 Progress Report #9, pp. 139-141.
34. Krugman S. Viral hepatitis type B: Prospects for active immunization. In: International Symposium on Viral Hepatitis, Milan, Dec. 1974.
Develop. biol. Standard. Vol. 30, Munich: S. Karger Basel, 1975, pp. VI; 363-367; the General Discussion can be found on pp. 375-379.
35. Purcell RH. Current understanding of hepatitis B virus infection and its implications for immunoprophylaxis. In: Antiviral Mechanisms: Perspectives in Virology IX, The Gustav Stern Symposium. New York: Academic Press, 1975 pp. 49-76.
36. Krugman S, Giles JP and Hammond J. Infectious hepatitis: Evidence for two distinctive clinical, epidemiological, and immunological
types of infection. JAMA 1967;200;5:366-373(96-103).
37. Shorter E. The Health Century: A companion to the PBS televisioneries. New York: Doubleday, 1987, pp. 67-69; 195-204. Maurice Hilleman
was interviewed by Edward Shorter on February 6, 1987. A copy of the audiotaped interview is held in the archives of the National Library
of Medicine, History Division, Washington, D.C.
38. Horowitz LG. Emerging Viruses: AIDS & Ebola, Nature, Accident or Intentional? Tetrahedron, LLC, 1996 p. 481.
39. Stricker RB and Elswood BF. Origin of AIDS (letter to the editor). The Lancet 1992;339:867.

Copyright c 1996, Leonard G. Horowitz. All rights reserved.

It’s due to Dr. Horowitz’s work that I first became aware of the issue, and since then, I have come to greatly respect his work. Thank you for reminding me, I will need to dig out “Emerging Viruses: AIDS and Ebola” for the tracker.

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post 17-June-2007 19:11
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1)When were the earliest case symptoms of AIDS… 2)When were the first studies of biochemical warfare?

Thank you.

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post 17-June-2007 21:06
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Biochemical warfare pre-dates AIDS by a considerable length of time, decades at least. From this page:


In an interview in 1948 between Heinrich Mueller, the former head of the Gestapo, and his CIA interrogator, Mueller reflected the same penny pinching cheapskate concern characteristic of fascists:

“If Stalin invades Europe … a little disease here and there would wipe out Stalin’s hoards and leave everything intact. Besides, a small bottle of germs is so much cheaper than an atom bomb, isn’t it? Why, you could hold more soldiers in your hand than Stalin could possibly command and you don’t have to feed them, clothe them or supply them with munitions. On the other hand, the threat of war … does wonders … for the economy.”19

Apparently, designing new kinds of bio-weapons for the DOD can be dangerous to a scientist’s health. More than 50 world-class scientists have been murdered since 2002 in the U.S. and the U.K. Almost all of them were microbiologists working on ethnic specific bio-weapons.